Liposarcoma is the most common retroperitoneal sarcoma and represents more than 15% of all sarcomas. Well differentiated liposarcoma is usually located in the extremities. We report the case of a patient with a giant retroperitoneal liposarcoma revealed by an abdominal mass.

Spontaneous rupture of the kidney consists of the atraumatic disruption of the renal parenchyma, followed by hemorrhage or leakage of urine into the perinephric space. A17 years old male known case of Acute Lymphoblastic Leukemia (ALL), referred to the urology department with right loin pain and fever for 3days. There was no history of trauma. Investigations revealed low hemoglobin, low platelets 18000 and urine analysis showed pus cells 14-18 cell/HPF and RBCs 2-4 /HPF. Renal functions tests and coagulation profi le were normal. CT urography showed bulky right kidney with mid pole laceration leaking of the contrast in the perinephric space. Conservative treatment was done (Double j insertion-ultrasound guided aspiration). Postoperatively he was generally well, afebrile, pain subside and repeated abdominal U/S showed no collection. Conservative management can be appropriate where clinical signs stabilize.

Transverse myelitis is an autoimmune demyelination disease of the spinal cord that can present with a myriad of symptoms ranging from altered sensations, weakness, loss of bowel control and so forth. There are approximately 1400 new cases reported each year in the United States, but this is likely an underestimation, without a gender bias or familial predisposition and a bimodal age distribution of onset (between 10-19 and 30-39) [1]. The majority of cases are idiopathic and so post-infectious is cited as the reason, although there are secondary causes such as being on the spectrum of multiple sclerosis, neurosarcoidosis, paraneoplastic syndromes and many others. This disease has a 5-10% chance of progressing to other demyelinating diseases, such as multiple sclerosis [2].

Familial hyperlipidemia is defi ned by abnormal levels of the following: low density lipoprotein cholesterol (LDL-C); high density lipoprotein cholesterol (HDL); triglycerides (TAG); or any combination of the three. Furthermore, while the disease is of a polygenic nature and at current time is poorly understood, it has been noted that the proteins apoB (needed for the LDL receptor and to aid in the removal of LDL from circulation) and apoE (which aids in the clearance of chylomicrons and very low density lipoprotein) present abnormalities in this illness [1]. With respect to apoB abnormalities, a Mendelian inheritance pattern has been observed in a number of cases despite the fact that familial hyperlipidemia is determined by interaction of multiple genes [2,3] and occurs in only 1-2% of the general population [4]. Anemia itself has numerous forms and causes, but the one that this paper will be focusing on will be iron defi ciency anemia. As this is a pediatric case, the thresholds will be for children 6 months to < 5 years of age, and are the same as those used by the World Health Organization: hemoglobin under 11g/dL and Ferritin <12 micrograms/L [5]. Iron defi ciency anemia is the most common form of anemia on the planet, ranging as high as 1 in 3 children less than 5 years of age [6].